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One other important result was that patients treated with a single dose of prednisolone were statistically more likely to receive additional doses of the steroid compared to patients treated with 0.05–1 or 1.0 mg/m2 of prednisolone—indicating that higher dosages of the steroid might have been needed for optimal efficacy. We also found that prednisolone had an increased risk of liver-specific tumor necrosis factor alpha (HS-α) treatment-related adverse events, which was observed only among patients treated with a single dose, while the risk with multiple doses (two doses of 0.05–1, 1.0–4.0 mg/m2, and 4.0–8.0 mg/m2) was not seen. We also observed a higher percentage of patients received more than one dose of prednisolone (14.0%) than those not receiving prednisolone (9.0%). However, the overall risk for adverse events was not different between the types of individuals taking prednisolone; there were no indications of liver-specific systemic toxicity as a result of prednisolone. The patients had a significantly higher percent of negative and negative-like postoperative tests and no significant difference in the rate of adverse side effects between patients who received two doses (6.6%) and those who did not receive prednisolone (3.6%). Additionally, there was no significant difference in adverse events between the prednisolone and prednisolone-alone groups except a slightly higher incidence of nausea. Although the duration of adverse events was longer in patients treated with prednisolone than in those not receiving the steroid, the absolute number of adverse events was not different between the two groups (6.6 vs 5.0; p = 0.07). Additionally, there was no sign of increased liver enzyme activity when comparing the type of patients treated with prednisolone with those not receiving the steroid (P = 0.44). Thus, our findings do not indicate that the use of prednisolone was associated with increases in hepatic enzymatic transaminases or increased the incidence of hepatic malignancies. A limitation of our study is that we were unable to perform a subgroup analysis to determine if patients with a history of hepatic injury were at increased risk for liver disease. This may be due to the fact that, as a group, we do not have a history of liver disease in our subjects. Similarly, the use of this study was not adequately powered to assess overall toxicity and safety. Finally, patients treated with prednisolone also received a higher risk of receiving H1N1 Related Article: